The new study identifies two previously unknown neuroprotective pathways that may work increasingly less well as organisms age, helping to explain the close association between aging and Alzheimer's at a molecular level. One pathway disassembles protein aggregates so they can't harm cells. The second, which likely is activated when the first is overtaxed, protects cells by converting small toxic aggregates into less toxic larger ones, perhaps so they can be stored until they can be disposed of properly.
by Stu Borman |
August 14, 2006